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Deposition of advanced glycation end products (AGE) and expression of the receptor for AGE in cardiovascular tissue of the diabetic rat.


Treatment of hypercholesteremic mice with recombinant IFN-γ also accelerates atherosclerotic plaque development [167], an effect that is reversed when mice receive the drug pentoxyfyllinv[sic], a potent Th1 blocker [155].
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